Persistent pain results in suffering and poor quality of life. In addition, the lack of proper control of persistent pain may lead to the development of physical and mental disabilities, which further devastate the patient’s quality of life. The main goal of our research activities is to identify pivotal mechanisms in neuronal processing of information induced by various persistent painful conditions, which could be used to develop new pain killers. Hence, our activities will ultimately improve patients’ quality of life.
@article{Tahim:2005:10.1016/j.neuroscience.2005.08.005,
author = {Tahim, AS and Sántha, P and Nagy, I},
doi = {10.1016/j.neuroscience.2005.08.005},
journal = {NEUROSCIENCE},
pages = {539--548},
title = {Inflammatory mediators convert anandamide into a potent activator of the vanilloid type 1 transient receptor potential receptor in nociceptive primary sensory neurons},
url = {http://dx.doi.org/10.1016/j.neuroscience.2005.08.005},
volume = {136},
year = {2005}
}
TY - JOUR
AU - Tahim,AS
AU - Sántha,P
AU - Nagy,I
DO - 10.1016/j.neuroscience.2005.08.005
EP - 548
PY - 2005///
SN - 0306-4522
SP - 539
TI - Inflammatory mediators convert anandamide into a potent activator of the vanilloid type 1 transient receptor potential receptor in nociceptive primary sensory neurons
T2 - NEUROSCIENCE
UR - http://dx.doi.org/10.1016/j.neuroscience.2005.08.005
UR - https://www.webofscience.com/api/gateway?GWVersion=2&SrcApp=PARTNER_APP&SrcAuth=LinksAMR&KeyUT=WOS:000233832600017&DestLinkType=FullRecord&DestApp=ALL_WOS&UsrCustomerID=a2bf6146997ec60c407a63945d4e92bb
VL - 136
ER -